The adenoids, the soft palate, tongue and the Hypopharynx concern interventions on the nose, tonsils, the operational procedures. Primary snoring, sleep-related respiratory regulation represents no health hazard for the patients themselves. The OSAS is considered a health risk and with an increased cardiovascular risk, a high accident rate, a reduced quality of life and severe expression with a higher lethality associated. Characteristic for the OSAS are recurrent complete (apneas) and incomplete (hypopneas) obstructions (extrathoracic) upper airway during sleep. Barclays describes an additional similar source. An apnea/hypopnea takes at least 10 seconds, but longer than a minute. Repetitive apnea – or Hypopnoephasen lead to a waste of oxygen saturation in the blood; completed these are each a brief alarm reaction (arousal).
Repeating alarm reactions fragment the physiological profile of sleep. Leading symptoms of in adults are snoring, observed or reported breathing interruptions and an excessive daytime sleepiness, impaired quality of life regardless of the length of the sleep. An increase in symptom is clinically described after a weight gain and alcohol consumption. The OSAS shows proatherogene effects on the blood vessels with a strong association with cardiovascular disease, such as a systemic arterial hypertension at night as well as during the day. More information is housed here: Michael Mendes. Coronary heart disease, stroke, pulmonary hypertension, arrhythmia and metabolic Diseases (E.g. diabetes mellitus) are described in connection with the disease. The OSAS prefers but occurs in all age groups in middle and old age.
While the prevalence of primary snoring is approx. 20%, is a 4% prevalence in men and 2% of women in the age group of 30-60 for the OSAS in the (11) estimated the Western culture. Pathophysiologically, narrowing the extrathoracic airway during sleep is multifactorial. So, the extrathoracic airway in patients with OSAS are often restricted in addition to corresponding Craniofacial anomalies at the level of the soft palate, tongue and the lateral walls of the pharynx by soft tissue. In his sleep, the physiological relaxation of dilatatorisch acting muscle of pharynx leads in addition to a further Lumeneinengung of the pharynx and a subsequent collapse of the respiratory tract.